Basic/Clinical Science Session

New Participants in AKI: Cellular Components, Signaling, and More

November 08, 2019 | 02:00 PM - 04:00 PM

Location: 207, Walter E. Washington Convention Center

Session Description

AKI is conventionally regarded as the result of abnormalities in and interplay among the epithelial cell, the endothelial and other vascular cells, and inflammatory processes, along with abnormalities in renal energetics. This session presents new mechanisms and participants in AKI. These include processes arising in specific cellular compartments (lymphangiogenesis), involving specific cells (neutrophil extracellular traps), involving signaling in specific cells (interstitial fibroblasts), and originating from unexpected metabolic pathways (fructose metabolism).

Learning Objective(s)

  • Describe the mechanisms of AKI
  • Explain new pathways in AKI

Learning Pathway(s)

  • Acute Kidney Injury


  • Subhashini Bolisetty, PhD
  • Ambra Pozzi, PhD


  • The Role of Lymphangiogenesis
    02:00 PM - 02:30 PM
    Abolfazl Zarjou, MD, PhD
  • The Role of Neutrophil Extracellular Traps
    02:30 PM - 03:00 PM
    William Brian Reeves, MD, FASN
  • The Role of Wnt/Beta-Catenin Signaling in Interstitial Fibroblasts
    03:00 PM - 03:30 PM
    Youhua Liu, PhD
  • The Role of the Polyol Pathway
    03:30 PM - 04:00 PM
    Miguel A. Lanaspa, PhD, DVM