New Participants in AKI: Cellular Components, Signaling, and More
November 08, 2019 | 02:00 PM - 04:00 PM
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Basic/Clinical Science Session
New Participants in AKI: Cellular Components, Signaling, and More
November 08, 2019 | 02:00 PM - 04:00 PM
Location: 207, Walter E. Washington Convention Center
Session Description
AKI is conventionally regarded as the result of abnormalities in and interplay among the epithelial cell, the endothelial and other vascular cells, and inflammatory processes, along with abnormalities in renal energetics. This session presents new mechanisms and participants in AKI. These include processes arising in specific cellular compartments (lymphangiogenesis), involving specific cells (neutrophil extracellular traps), involving signaling in specific cells (interstitial fibroblasts), and originating from unexpected metabolic pathways (fructose metabolism).
Learning Objective(s)
- Describe the mechanisms of AKI
- Explain new pathways in AKI
Learning Pathway(s)
Moderators
- Subhashini Bolisetty, PhD
- Ambra Pozzi, PhD
Presentations
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The Role of Lymphangiogenesis
02:00 PM - 02:30 PM
Abolfazl Zarjou, MD, PhD
The Role of Lymphangiogenesis
November 08, 2019 | 02:00 PM - 02:30 PM
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The Role of Neutrophil Extracellular Traps
02:30 PM - 03:00 PM
William Brian Reeves, MD, FASN
William Brian Reeves, MD, FASN
The Role of Neutrophil Extracellular Traps
November 08, 2019 | 02:30 PM - 03:00 PM
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The Role of Wnt/Beta-Catenin Signaling in Interstitial Fibroblasts
03:00 PM - 03:30 PM
Youhua Liu, PhD
The Role of Wnt/Beta-Catenin Signaling in Interstitial Fibroblasts
November 08, 2019 | 03:00 PM - 03:30 PM
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The Role of the Polyol Pathway
03:30 PM - 04:00 PM
Miguel A. Lanaspa, PhD, DVM
Miguel A. Lanaspa, PhD, DVM
The Role of the Polyol Pathway
November 08, 2019 | 03:30 PM - 04:00 PM
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