ASN's Mission

To create a world without kidney diseases, the ASN Alliance for Kidney Health elevates care by educating and informing, driving breakthroughs and innovation, and advocating for policies that create transformative changes in kidney medicine throughout the world.

learn more

Contact ASN

1401 H St, NW, Ste 900, Washington, DC 20005

email@asn-online.org

202-640-4660

The Latest on X

Kidney Week

Abstract: SA-PO0306

Intracellular Lipid Accumulation and Injury in Diabetic Kidney Disease and the Effect of Newly Synthesized Cyclodextrin

Session Information

Category: Diabetic Kidney Disease

  • 701 Diabetic Kidney Disease: Basic

Authors

  • Ryu, Jaejin, Yonsei University College of Medicine, Seodaemun-gu, Seoul, Korea (the Republic of)
  • Kang, Dong Hoon, National Health Insurance Service Ilsan Hospital, Goyang-si, Gyeonggi-do, Korea (the Republic of)
  • Nam, Boyoung, Yonsei University College of Medicine, Seodaemun-gu, Seoul, Korea (the Republic of)
  • Yoo, Tae-Hyun, Yonsei University College of Medicine, Seodaemun-gu, Seoul, Korea (the Republic of)

Group or Team Name

  • Institute of Kidney Disease Research.
Background

Lipid dysmetabolism is one of the key pathogenic mechanisms and a potential therapeutic target in diabetic kidney disease. Cyclodextrins have been used to modulate cholesterol metabolism due to their ability to bind to and solubilize cholesterol; however, their non-specific cholesterol depletion induces cytotoxicity, limiting their therapeutic use.

Methods

In this study, we evaluated the therapeutic potential of a newly synthesized cyclodextrin (NCD). In vivo experiments were conducted in db/db and db/m mice administered with vehicle or NCD at doses of 200, 500, or 2000 mg/kg. In vitro, mouse mesangial cells were treated with 50ug of fluorescent labeled cholesterol crystals followed by hydroxypropyl beta-cyclodextrin (HPβCD) or NCD.

Results

In vivo, db/db mice exhibited significant elevation of serum creatinine, proteinuria, and kidney cholesterol contents compared to db/m controls. Treatment with NCD demonstrated kidney protective effects, with dose-dependent reductions in serum creatinine and proteinuria, as well as markedly reduced kidney cholesterol contents. In vitro, NCD effectively promoted cholesterol efflux in mesangial cells. Compared to hydroxypropyl beta-cyclodextrin (HPβCD), NCD showed excellent cell viability and minimal toxicity.

Conclusion

Our findings suggest that NCD may serve as a promising therapeutic candidate in diabetic kidney disease.

Funding

  • Government Support – Non-U.S.

Digital Object Identifier (DOI)