ASN's Mission

To create a world without kidney diseases, the ASN Alliance for Kidney Health elevates care by educating and informing, driving breakthroughs and innovation, and advocating for policies that create transformative changes in kidney medicine throughout the world.

learn more

Contact ASN

1401 H St, NW, Ste 900, Washington, DC 20005

email@asn-online.org

202-640-4660

The Latest on X

Kidney Week

Abstract: FR-PO1024

Urinary mRNA Biomarkers for the Noninvasive Diagnosis of Calcineurin Inhibitor Toxicity in Kidney Transplant Recipients

Session Information

Category: Transplantation

  • 2102 Transplantation: Clinical

Authors

  • Lee, Yu ho, CHA University Bundang Medical Center, Seongnam-si, Gyeonggi-do, Korea (the Republic of)
  • Baek, Jihyun, CHA University Bundang Medical Center, Seongnam-si, Gyeonggi-do, Korea (the Republic of)
  • Seo, Jung-Woo, Kyung Hee University, Dongdaemun-gu, Seoul, Korea (the Republic of)
  • Lee, So-young, CHA University Bundang Medical Center, Seongnam-si, Gyeonggi-do, Korea (the Republic of)
  • Jeong, Hye Yun, CHA University Bundang Medical Center, Seongnam-si, Gyeonggi-do, Korea (the Republic of)
  • Lee, Sangho, Kyung Hee University, Dongdaemun-gu, Seoul, Korea (the Republic of)
  • Hwang, Hyeon Seok, Kyung Hee University, Dongdaemun-gu, Seoul, Korea (the Republic of)
Background

Calcineurin inhibitor (CNI) toxicity is an important cause of graft dysfunction in kidney transplant recipients, yet distinguishing it from acute rejection (AR) and acute tubular necrosis (ATN) remains challenging. This study explores urinary mRNA biomarkers as a non-invasive tool to accurately identify CNI toxicity.

Methods

We retrospectively enrolled 110 kidney transplant recipients and classified them into four groups: stable graft function (n = 35), CNI toxicity (n = 25), acute rejection (n = 30), and acute tubular necrosis (n = 20). Biomarker candidates were identified from the GEO database. Urinary mRNA was extracted from cell pellets, reverse-transcribed, and quantified using real-time PCR.

Results

Four transcripts (LTF, NNMT, WFDC2, and HIF1A) were selected as candidate biomarkers. Urinary LTF, NNMT, and HIF1A levels were significantly lower in CNI toxicity group than in AR group, and NNMT and HIF1A were also lower than in ATN group. WFDC2 levels showed no significant differences across groups. Among single markers, urinary HIF1A best distinguished CNI toxicity from AR and ATN (area under the curve [AUC] = 0.845, p<0.001). Combining LTF, NNMT, and HIF1A further improved diagnostic accuracy (AUC = 0.867, p<0.001).

Conclusion

Urinary mRNA levels of LTF, NNMT, and HIF1A effectively distinguished CNI toxicity from AR and ATN. These findings support their potential as non-invasive diagnostic tools for graft dysfunction in kidney transplant recipients.

Digital Object Identifier (DOI)