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Abstract: SA-PO0496

Dapagliflozin Ameliorates Obesity-Induced Renal Sodium Handling Dysfunction via Inhibition of IL-6/STAT3 Signaling

Session Information

Category: Fluid, Electrolytes, and Acid-Base Disorders

  • 1101 Fluid, Electrolyte, and Acid-Base Disorders: Basic

Authors

  • Zhao, Shasha, Department of Biotechnology & Engineering, College of Environmental & Chemical Engineering, Yanshan University, Qinhuangdao, China
  • Cai, Jiahui, Department of Biotechnology & Engineering, College of Environmental & Chemical Engineering, Yanshan University, Qinhuangdao, China
  • Sun, Feifei, Department of Biotechnology & Engineering, College of Environmental & Chemical Engineering, Yanshan University, Qinhuangdao, China
  • Sun, Yunbo, Department of Biotechnology & Engineering, College of Environmental & Chemical Engineering, Yanshan University, Qinhuangdao, China
  • Pan, Qiaoyun, Department of Biotechnology & Engineering, College of Environmental & Chemical Engineering, Yanshan University, Qinhuangdao, China
  • Yang, Feng, Department of Biotechnology & Engineering, College of Environmental & Chemical Engineering, Yanshan University, Qinhuangdao, China
  • Tan, Runyan, Department of Biotechnology & Engineering, College of Environmental & Chemical Engineering, Yanshan University, Qinhuangdao, China
  • Dong, Fengxiao, Department of Biotechnology & Engineering, College of Environmental & Chemical Engineering, Yanshan University, Qinhuangdao, China
  • Liu, Weiping, First Hospital of Qinhuangdao, Qinhuangdao, Hebei, China
  • Pierre, Sandrine V., Marshall Institute for Interdisciplinary Research, Marshall University, Huntington, West Virginia, United States
  • Yan, Yanling, Key Labs Nanobiotech & Applied Chemistry, Department of Biotechnology & Engineering, College of Environmental & Chemical Engineering, Yanshan University, Qinhuangdao, China

Group or Team Name

  • S&T Program of Hebei and Hebei Provincial Department of Human Resources and Social Security.
Background

The sodium-glucose cotransporter 2 (SGLT-2) inhibitor dapagliflozin (Dapa) exhibits renal protective properties in metabolic disorders. However, the underlying mechanisms remain incompletely understood. This study investigates the role of IL-6/STAT3 signaling in obesity-induced impairment of renal sodium handling and the therapeutic potential of Dapa.

Methods

Diet-induced obese (DIO) C57BL/6J mice were used to assess the effects of Dapa on renal IL-6/Stat3 signaling and sodium excretion in response to a high-salt challenge. In vitro mechanistic studies were performed in human (HK-2) and pig (LLC-PK1) renal proximal tubular cells. The phosphorylation status of STAT3 and ERK1/2 following IL-6 stimulation, with or without Dapa or the SRC inhibitor PP2, was analyzed by immunoblotting.

Results

IL-6/Stat3 signaling was markedly activated in the renal cortex tissues of DIO mice. Obese mice exhibited significantly impaired sodium excretion under high-salt conditions compared to lean controls. Dapa treatment suppressed renal IL-6/Stat3 activation and restored natriuretic responsiveness. In HK-2 and LLC-PK1 cells, Dapa inhibited IL-6-induced phosphorylation of ERK1/2 and Stat3. Similarly, the SRC kinase inhibitor PP2 attenuated IL-6-mediated ERK1/2 and Stat3 activation (Figure 1), suggesting a shared upstream pathway.

Conclusion

These findings highlight a pivotal role of IL-6/STAT3 signaling in mediating obesity-related renal sodium handling defects. Dapagliflozin confers renoprotection by suppressing this pro-inflammatory signaling cascade. Targeting IL-6/STAT3 may represent a novel therapeutic approach for obesity-associated renal dysfunction.

Figure 1. The impact of dapagliflozin on IL-6/Stat3 signaling

Funding

  • NIDDK Support

Digital Object Identifier (DOI)