Posters
Glomerular Diseases: Omics
October 22, 2026 | 10:00 AM - 12:00 PM
Location: Exhibit Hall A, Convention Center
Session Description
Poster Session
Posters
Urinary Single-Cell Transcriptomics Reveals Parietal Epithelial Cell State Plasticity in FSGS
IFNα-Induced CD169+ Macrophages Drive Inflammation via the CCL2/NLRP3 Axis in IgAN: Transcriptomics and Functional Characterization
Soluble CD93 Mediates Podocyte Injury Through Activation of the TGF-β Pathway in Idiopathic Nephrotic Syndrome
Spatial Transcriptomics Reveals Glomerular Molecular Boundary Associated with Treatment Response in Patients with FSGS
Proteomic Profiling Reveals Complement Activation and Extracellular Matrix Remodeling in Renal Amyloidosis
Single Glomerulus Proteomics: A GLIMPSE into Crescentic Glomerulonephritis
Global Transcriptomic Responses in Precision-Cut Kidney Slice Cultures
Intrarenal Activation of Complement and SPP1 Pathways in Staphylococcus Infection-Associated Glomerulonephritis (SAGN): A Single-Cell Spatial Transcriptomic Study
SRSF3 and EPB41L5 Isoform Switching Reveals a Conserved Splicing Program of Podocyte Injury in FSGS
Multilayered Affinity Proteomics Reveal the Protein Interaction Network of IgAN
Integrated Spatial Multi-Omic Profiling Reveals Spatial Heterogeneity in C3 Glomerulonephritis
Multispecies, Single-Cell, Multi-Omic Analysis Reveals the Mechanism of Podocyte Species Conserved Molecule GOLIM4
Single-Cell Transcriptome Sequencing Reveals That Periglomerular Macrophages Aggravate Podocyte Injury in Minimal Change Disease
Integrative Small RNA and Transcriptomic Profiling of Formalin-Fixed Paraffin Embedded Kidney Biopsies from a Lupus Nephritis Inception Cohort Reveals Novel and Clinically Relevant MicroRNA Signatures
Untargeted Single Glomerulus Proteomics Reveals Sex-Specific Aging States
Multi-Omic Approach to Define Multimolecular Plasma Signatures of Active and Recurrent FSGS After Kidney Transplantation
Spatial Transcriptomics (ST) in Kidney Biopsies of Patients with IgAN
Cross-Disease Urinary Epiproteomics Identifies Reduced Collagen Oxidation as a Generalizable Mechanistic Biomarker of Kidney Damage
Integrated Peripheral and Spatial Single-Cell Multi-Omics Reveals Immune Remodeling in Minimal Change Disease
Antifibrinolytic Transcriptomic Signature in Membranous Nephropathy: Insights from Glomerular Gene Expression Analysis
Urinary Complement Profiling as a Tool for Disease Activity in Glomerular Disorders
Immunological Characterization of IgAN Using Proteomic Analysis of Laser Capture Microdissected Palatine Tonsils
Serum Label-Free Quantitative Acetylated Proteomic Analysis in Patients with Idiopathic Membranous Nephropathy
Quantification of Podocyte Foot Process Effacement in Mouse Models of CKD Using Direct Stochastic Optical Reconstruction Microscopy
Podocyte Retroelement Activation Links Albuminuria to Depression-Related Neuroimmune Changes Through Blood-Brain Barrier Dysfunction and Th1 Skewed CD4 T-Cell Brain Entry
Podocytic Injury Alters Circadian Gene Expression in the Kidney Glomeruli
Characterization of IgA, IgA-Immune Complexes and Complement in IgAN Using Laser Microdissection and Mass Spectrometry
Pervasive Alternative Splicing, Alternative Polyadenylation, and Nonsense-Mediated mRNA Decay (NMD)-Coupled Isoform Switching Across Multispecies Models of Glomerular and Podocyte Disease
Fosl2(+) Parietal Epithelial Cells Seed Myofibroblast Populations in Crescentic Glomerulonephritis
Computationally Derived Tubular Morphometric Signatures Define Transcriptionally Distinct States of Tubular Injury in FSGS/Minimal Change Disease (MCD)
DEAD-Box Helicase 41 (DDX41) as a Candidate Driver of Podocyte Injury
TEAD1 Activity Links Lipid Membrane Composition to FSGS Progression
Weighted Gene Coexpression Network Analysis (WGCNA) Identifies GAS6/AXL and Hedgehog/GLI1 Signaling as Associated with Fibrotic Progression in ANCA-Associated Vasculitis Glomerulonephritis
Potential IgAN Biomarkers from the Glycine, Serine, and Threonine Metabolism Pathway
Proteinuria Reduction by Methylprednisolone, Pioglitazone, and Mifepristone Is Mediated by Shared Gene Expression and Pathway Remodeling in Experimental Nephrotic Syndrome
Deconvolution Analysis and Animal Experiments Reveal the Relationship Between the NLRP3 Inflammasome Pathway and Crescentic Glomerulonephritis
N-Glycosylation Patterns of Urinary Clusterin Determine the Severity of Diabetic Kidney Disease
Cross-Phenotype Genome-Wide Association Study Identifies Shared Genetic Susceptibility Across Glomerular Disorders