Oral Abstract Session
Glomerular Diseases: Novel Mechanisms of Injury
October 22, 2026 | 04:30 PM - 06:00 PM
Location: Room 401, Convention Center
Session Description
Oral Abstract Session
Learning Pathway(s)
- Glomerular Diseases
Moderators
Presentations
Macrophage Ontogeny Dictates the Spatiotemporal Accumulation and Function of Kidney-Resident Macrophages in Crescentic Glomerulonephritis (GN)
Eosinophils-Mediated Protection Against Lupus Nephritis: Characterization of the Mechanisms and Demonstrating the Potential of IL-5 as a Novel Therapeutic Approach
Intrarenal CCL2 and CCR2 Activation Profiles Are Linked to Noninvasive Urinary Biomarkers Associated with Outcomes in Patients with FSGS
Kidney and Immune STAT3 Targets Drive Severity in FSGS and Minimal Change Disease: A NEPTUNE/CureGN Study
Single-Nucleus Transcriptomic Landscape of IgAN Reveals Severity-Linked Glomerular Cell-State Transitions and a Panglomerular Molecular Signature
Integrated In Vitro, Organ-on-a-Chip, and In Vivo Evidence of Microplastic-Induced Kidney Dysfunction
CD11b Activation Ameliorates Lupus Nephritis by Reducing Myeloid Cell Infiltration, Including rs1143679 Carriers
Breakdown of Oral Tolerance to Gut Bacteria-Derived Galactose-Deficient IgA1 Drives IgAN
Kidney CCL20-CCR6 Axis Drives Th17 Recruitment in Immune Complex-Mediated Glomerulonephritis