ASN's Mission

To create a world without kidney diseases, the ASN Alliance for Kidney Health elevates care by educating and informing, driving breakthroughs and innovation, and advocating for policies that create transformative changes in kidney medicine throughout the world.

learn more

Contact ASN

1401 H St, NW, Ste 900, Washington, DC 20005

email@asn-online.org

202-640-4660

The Latest on X

Kidney Week

Please note that you are viewing an archived section from 2019 and some content may be unavailable. To unlock all content for 2019, please visit the archives.

Abstract: SA-PO792

Dietary Modification Improves FGF-23, KLOTHO, PTH, and Serum Phosphorous Levels in CKD Stages 1 and 2

Session Information

Category: Health Maintenance, Nutrition, and Metabolism

  • 1300 Health Maintenance, Nutrition, and Metabolism

Author

  • Saxena, Anita, Sanjay Gandhi Post Graduate Institute of Medical Sciences, Lucknow, India

Group or Team Name

  • SGPGI Renal Nutrition Group
Background


Dietary phosphorus restriction is a potential therapy for improving cardiovascular outcomes.
Aim: To examine effect of dietary counselling and dietary modifications on FGF-23, klotho, PTH levels and phosphorous in CKD Stage 1 and Stage 2 patients.

Methods


100 subjects aged 35.37 ± 10.98 years with eGFR 83.55+16.53 ml/minute and BMI 24.74 ±2.18 were recruited in the study. 24 hour dietary recall was taken at baseline, Based on dietary phosphorous intake patients were divided into two groups. Group 1: low phosphorous diet and Group 2: high. phosphorous diet. Controls were 30 healthy subjects, aged 43.83±10.11 ml/min with eGFR 126.11+10.26 ml/min and BMI 24.06 ±2.09. FGF-23, soluble α-Klotho, iPTH were measured (ELISA)

Results

The independent t test showed that iPTH, serum creatinine, FGF-23, serum phosphorous, total cholesterol and VLDL were significantly higher in CKD patients compared to controls(p=0.000),. Hemoglobin (p=0.046) , vitamin D(p=0.008) , klotho (p=0.000) and HDL (p=0.000) were lower in CKD patients compared to controls (n=30).
GFR showed a positive significant correlation with klotho (r=0.696,p=0.000) and a negative significant correlation with serum phosphorous (r=-0.494,p=0.000), iPTH (r=-0.751, p=0.000), FGF-23 (r=-0.638,p=0.000), urinary phosphorous (r=-0.476,p=0.000) and dietary phosphorous intake (r=-0.678,p=0.000) .
Urinary phosphorous showed a significant positive correlation with dietary phosphorous (r=0.488, p=0.000)
After dietary counselling and diet modifications in group 2, dietary phosphorous decreased from 1384.74±117.32 to 1027.69±101.39 (p=0.008; serum phosphorus 3.89±0.57 to3.59±0.63 p 0.008), FGF-23 (169.80±50.96 to 159.45±58.66; p 0.023), klotho (351.77±134.88 to 316.83±167.25 p 0.037) and iPTH decreased from 88.48±10.55 to 85.49±15.25; p 0.033. There was decline in protein intake from 0.67±0.130.to 60±0.11g/kg/d p 0.010. FGF-23 showed a strong significant negative correlation with klotho (r=0.754,p=0.000) i.e. as the levels of FGF-23 decreases, the levels of klotho increases after the diet modification in group 2 when they were advised to be strictly on a low phosphorous diet.

Conclusion


Both diet counselling and diet modification can bring down levels of FGF-23,Klotho, iPTH, serum phosphorous, urinary phosphorous which can be instrumental in slowing the progression of CKD and preventing cardiovascular disease.