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Abstract: SA-PO496

TNF-α Inhibition Protects Against Renal Tubulointerstitial Injury Associated with Suppressing NLRP3 Inflammasome in Diabetic Rats

Session Information

Category: Diabetic Kidney Disease

  • 601 Diabetic Kidney Disease: Basic

Authors

  • Wang, Feng, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, Shanghai, China
  • Yin, Jianyong, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, Shanghai, China
  • Zhao, Ting, Shanghai Sixth People''s Hospital, Shanghai, China
  • Kong, Yiwei, Medical College of Wisconsin, Milwaukee, Wisconsin, United States
  • Liu, Xuanchen, Shanghai Jiao Tong University Afflicted Sixth People's Hospital, Shanghai, SHANGHAI, China
Background

Tubular injury exerts a pivotal role in the development of diabetic nephropathy (DN), but current therapies used to treat DN do not combat tubular injury. This study was conducted to investigate whether TNF-α inhibition protected against tubular injury in diabetic rats and examine the associated mechanisms.

Methods

Kidney biopsy tissues, taken from twelve patients with DN and five control subjects, were analyzed. STZ-induced diabetic rats were treated with a TNF-α inhibitor (Humira) for twelve weeks. Renal function, albuminuria, histological injury, renal TNF-α mRNA, and NLRP3 inflammasome was assessed.

Results

Diabetic patients with tubulointerstitial injury presented with higher renal tubular expression of TNF-α mRNA and NLRP3 inflammasome. TNF-α inhibition reduced albuminuria, glomerular injury, and tubular injury in STZ-induced diabetic rats. Importantly, TNF-α inhibition significantly reduced NLRP3 inflammasome in tubules. Moreover, TNF-α inhibition decreased expression of tubular IL-6 and IL-17A, which could activate NLRP3 inflammasome.

Conclusion

TNF-α inhibitor can protect against tubulointerstitial injury associated with suppressing NLRP3 inflammasome in diabetic rats.